visu nelasīju, bet pirmīt bija pieminēts lietuvēns...halucinācijas ir normāla parādība miega paralīzes laikā. lasiet un guliet mierīgi, jo tie toreiz nebija spoki, bet pazīstams smadzeņu stāvoklis.
Hypnagogic and hypnopompic hallucinations are symptoms commonly experienced during episodes of sleep paralysis. Some scientists have proposed this condition as an explanation for reports of alien abductions and ghostly encounters. A study by Susan Blackmore and Marcus Cox (the Blackmore-Cox study) of the University of the West of England supports the suggestion that reports of alien abductions are related to sleep paralysis rather than to temporal lobe lability. There are three main types of these hallucinations that can be linked to pathologic neurophysiology. These include the belief that there is an intruder in the room, the incubus, and vestibular motor sensations.
Many people that experience sleep paralysis are struck with a deep sense of terror, because they sense a menacing presence in the room while paralyzed—hereafter referred to as the intruder. This phenomenon is believed to be the result of a hyper vigilant state created in the midbrain. More specifically, the emergency response activates in the brain when individuals wake up paralyzed and feel vulnerable to attack. This helplessness can intensify the effects of the threat response well above the level typical to normal dreams; this could explain why hallucinations during sleep paralysis are so vivid. Normally the threat activated vigilance system is a protective mechanism the body uses to differentiate between dangerous situations and determine whether the fear response is appropriate. This threat vigilance system is evolutionarily biased to interpret ambiguous stimuli as dangerous, because "erring on the side of caution" increases survival chances. This could explain why those who experience sleep paralysis generally believe the presence they sense is evil. The amygdala is heavily involved in the threat activation response mechanism, which is implicated in both intruder and incubus SP hallucinations. The specific pathway the threat-activated vigilance system acts through is not perfectly understood. It is believed that either the thalamus receives sensory information and sends it on the amygdala, which regulates emotional experience—or that the amygdaloid complex, anterior cingulate, and the structures in the pontine tegmentum interact to create the hallucination. It is also highly possible that SP hallucinations could result from a combination of these. The anterior cingulate has an extensive array of cortical connections to other cortical area, which lets it integrate the different sensations and emotions we experience. The amygdaloid complex helps us interpret emotional experience and act appropriately. Most importantly, it helps us direct our attention to the most pertinent stimuli in a potentially dangerous situation and act appropriately. Proper amygdaloid complex function requires input from the thalamus. This creates a thalamoamygdala pathway capable of bypassing intense scrutiny of incoming stimuli, which allows for quick responses in a potentially life-threatening situation.
7682
